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There is much evidence to support that AAG is a disorder of synaptic transmission caused by specific antibodies against the α3 subunit of the nicotinic acetylcholine receptor (nAChR) at the autonomic ganglia level. 1 However, authors of sudomotor function studies have hypothesized that there is a postganglionic autonomic neuropathy, 2 which has been recently demonstrated on sural nerve biopsy findings. 3 Herein, we report a case of chronic AAG showing postganglionic autonomic and somatic ...

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